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SAC vs Calcium Supplements

By | Resources, SAC

SAC vs Calcium Supplements

Americans today are facing a serious health issue: More than 75% of Americans are calcium deficient, setting ourselves up for a major health breakdown.

The stomach needs a strongly acidic environment for calcium absorption from food or supplements. However, people over the age of 60 produce only 1/4 of the stomach acid they did when they were 20, leading to poor absorption of calcium.

Most of what is absorbed doesn’t end up in the bones, due to lack of exercise and a more sedentary life style.

Even with a plethora of calcium supplements sold in America, incidences of osteoporosis are still on the increase. Why?

There are many problems with traditional calcium supplements since their calcium absorption rates are usually too low to be of use. Sources – such as from coral, plants or algae, may vary in quality, but the problem is that absorbed calcium enters our blood vessels as inactive protein-bound calcium which our body cannot utilize directly to build up bones.

Protein calcium is only utilized after extensive exercise which most elderly Americans do not engage in. Even worse, high calcium intake in this protein-bound form develops unpleasant effects such as acid rebound and mineral imbalance, to name a few.

For these reasons, many trained medical professionals discourage the use of calcium supplements.

Building the Bone Density

Almost all your body’s calcium is stored in bone, but the tiny amount that circulates in your bloodstream is disproportionately vital to normal physiology.

About half of this circulating calcium (50%) is “ionized”, which means it carries electrical charges.

Ionized calcium (Ca2+) is the only physiologically active form that can be recognized by our body and absorbed in our bones.

Ionized calcium in the blood is so vital that the body cannot permit it to fluctuate. Therefore, even a slight increase in the concentration of ionized calcium in the blood triggers the bone-building process to take excess calcium into bones.

Utilizing this process is by far the most effective and safe way to build bone density since it follows the body’s natural bone building mechanism.*

Triggering Bone Formulation

When the calcium ion concentration rises even slightly, the thyroid gland immediately increases the secretion of calcitonin into the blood, which removes calcium from the blood plasma and deposits it as new bone.

The above process is both natural and safe, but there has simply been no way to add ionized calcium into our blood serum directly to initiate the bone formation process… until today.

The revolutionary SAC Formulation Technology has made possible what other calcium supplements and prescription drugs could not achieve. Supporting and sustaining the natural bone-building process through the flash of calcium ions.*

Bone Density Clinical Results

By | Resources, SAC

Bone Density Clinical Results

How the Measurement is Taken

The following clinical results are typical of patients taking SAC calcium daily. The results show the significant improvement in a relatively short time period. It is measured with FDA approved Omnipath Axial Transmission Ultrasound Technology by Beammed. Omnipath™ is a unique, patented, proprietary axial transmission technology based on the measurement of the speed of ultrasonic waves propagating along the bone.

How to Read Graphs

Blue dot represents initial bone mineral density measurement (BMD).
Red dot is the last measurement.

T-Score Range

-1.0 or higher indicates that BMD is Normal (green)
Between -1.0 and -2.5 indicates Osteopenia (yellow).
-2.5 or lower indicates Osteoporosis (red).
0 means your BMD is equal to the norm for a healthy young adult.

SAC Rebuilds Jaw Bones

Tooth Extraction

When an adult tooth is removed and not replaced, jawbone deterioration may occur. Natural teeth are embedded in the jawbone, and stimulate the jawbone through activities such as chewing and biting. When teeth are missing, the alveolar bone, or the portion of the jawbone that anchors the teeth in the mouth, no longer receives the necessary stimulation, and begins to break down, or resorb. The body no longer uses or “needs” the jawbone, so it deteriorates and goes away.

The rate the bone deteriorates, as well as the amount of bone loss that occurs, varies greatly among individuals. However, most lost occurs within the first eighteen months following the extraction, and continues throughout life.

X-ray A and B shows bone loss around extracted tooth. With SAC treatment for 10 months, 60-year-old male shows rapid building up of jaw bone even where a tooth is extracted.

Periodontal Disease:

Periodontal diseases are ongoing infections of the gums that gradually destroy the support of your natural teeth. Periodontal disease affects one or more of the periodontal tissues: alveolar bone, periodontal ligament, cementum, or gingiva. While there are many diseases which affect the tooth-supporting structures, plaque-induced inflammatory lesions make up the majority of periodontal issues, and are divided into two categories: gingivitis and periodontitis. While gingivitis, the less serious of the diseases, may never progress into periodontitis, it always precedes periodontitis.

Periodontitis is affected by bacteria that adhere to the tooth’s surface, along with an overly aggressive immune response to these bacteria. If gingivitis progresses into periodontitis, the supporting gum tissue and bone that holds teeth in place deteriorates. The progressive loss of this bone, the alveolar, can lead to loosening and subsequent loss of teeth.

X-ray C shows the teeth of 45-year-old male with jaw bone loss around his teeth. With SAC treatment, we observe rapid build up of jaw bone supporting teeth.

Osteonecrosis:

Osteonecrosis is a condition that occurs when there is loss of blood to the bone. Because bone is living tissue that requires blood, an interruption to the blood supply causes bone to die. If not stopped, this process eventually causes the bone to collapse. Osteonecrosis most commonly occurs in the hip. Other common sites are the shoulder, knees, shoulder, and ankles.

X-Ray D shows the leg of 10 year-old girl with an onset of bone necrosis. With SAC treatment, necrosis disappeared to her doctor’s dismay, and she is healthy today. Click on the x-ray to see more pictures.

SAC and Osteoporosis

By | SAC, Uncategorized

SAC and Osteoporosis

Bone is living, growing tissue. Throughout life, our bodies are breaking down old bone cells and rebuilding new bones in a continuous cycle (bone remodeling). We gain bone by building more bone cells than we lose. After about age 40, however, this balance is typically reversed, with bone loss occurring at a much faster rate than is replaceable, leaving our bones brittle and leading to osteoporosis with increased risks of fracture, particularly of the hip, spine, wrist, and shoulder.

Osteoblasts and osteoclasts are types of cells the human body uses to repair broken bones. Osteoclasts break down old bone tissue allowing osteoblasts to replace it with new material. Together, these cells facilitate bone mending and bone growth. However, as we age osteoblast slows down and bone mineral density continues to decrease, making our bones more susceptible to fracture.

Osteoporosis weakens bones

Losing more than 25% of bone mass is considered osteoporosis. In the US, 1/3 of women and 1/4 of men have the disease, with the figure increasing each year. What may be the cause of all this?*

The 4 Functions of SAC Calcium

SAC as Hormone Regulator

Bone metabolism is controlled by the interaction of a number of hormones.

Hormonal imbalance can wreak havoc on bone metabolism. SAC assures that calcium will directly reach the bone and will not be deposited in the kidneys and blood vessels.*

SAC as Calcium Navigator

Inactive calcium molecules such as protein-bound calcium do not know where to go.

SAC navigates calcium to reach its ultimate destination – the bone. SAC calcium will not be deposited in the wrong places such as the kidneys or blood vessels.*

SAC as Calcium Activator

About 50% of the calcium in our blood is inactive.

SAC’s ionization process activates inactive calcium for use by the body.*

SAC as Bone Mineral Builder

Adequate SAC intake, as part of a healthful diet, along with physical activity, may reduce the risk of osteoporosis in life.

Serious Side Effects of Prescription Drugs

Physicians typically recommend prescription medications for the treatment of osteoporosis, but these drugs are notorious for their dangerous side effects. These drugs are designed to “increase” bone density by retaining dead bone mass through the inhibition of osteoclasts, the cells that are responsible for natural bone resorption, which provides calcium for our body’s needs. Our body takes calcium from bones – even at the risk of bone fractures, because of calcium’s urgent and crucial role in healthy cell replication, neurotransmission, heart functions, and endocrine functions.

Disrupting essential bone metabolism may lead to serious health hazards.

This is why there are so many reports that osteoporosis drugs actually make bones more brittle and that people who are treated with these drugs tend to suffer more bone fractures.*

Two Categories of Prescription Drugs

Anti-resorptive Drugs

Drugs that slows bone loss

Antiresorptive therapies are used to increase bone strength by slowing or stopping osteoclast in individuals with osteoporosis and include five principal classes of agents: bisphosphonates, estrogens, selective estrogen receptor modulators (SERMs), calcitonin and monoclonal antibodies such as denosumab.

Bisphosphonates are the most commonly prescribed antiresorptive medications and remain first-line treatment for osteoporosis. Bisphosphonates are adsorbed into the mineralized surface of bone and are internalized by osteoclasts, interfering with biochemical processes involved in bone resorption; they also induce apoptosis of osteoclasts.

Denosumab, the first biologic introduced for osteoporosis treatment, is a fully human monoclonal RANKL antibody, and by binding to RANKL, it prevents the binding of RANKL to RANK; this leads to inhibition of osteoclast activation and function.

Anabolic Drugs

Drugs that increase the rate of bone formation

Trailing the development of antiresorptives for osteoporosis is the development of anabolic agents designed to increase bone mineral density (BMD) by stimulating bone formation, osteoblast. Sodium fluoride was a promising anabolic agent for the treatment of postmenopausal osteoporosis, but it was found to increase the risk of nonvertebral fractures despite dramatic increases in BMD and is not approved by FDA in the USA. GH (growth hormone) has also been the object of interest as an anabolic agent for the skeleton. PTH (parathyroid hormone), long known to have anabolic potential was “rediscovered” about 15 yrs ago, but the bone-building effect was not very impressive (only 6% increase) and also came with many side effects.

The statins, cornerstones of lipid-lowering therapy, have also recently been revisited as potentially important skeletal anabolic agents, but we are too familiar with the side effects of statins.

The Clinical Results of SAC

Human Trial Result

This indicates real bone mineral density (BMD) increase of 49 year-old male since taking SAC calcium. Measurement shows a significant increase of BMD (T-score of 1.5) over a period of 6 months. BMD is measured by FDA approved ultrasound bone densitometer by BeamMed.

Animal Trial Result

Lab Animal Test 2011: 27(4), 301-307

Effects of SAC Calcium on bone turnover and calcium balance in ovariectomized rats.

Results reveal that the BMD of rats with induced osteoporosis has returned to normal with SAC intake. Please click the link below for the full research paper.

View Research StudyView Research Study
Group
BMD
Sham (Control)
0.2276 ± 0.011 a
OVX (Osteoporosis)
0.1965 ± 0.012 b
OVX + SAC
0.2276 ± 0.012 a